Advances in lipid research. / Volume 14 by Rodolfo Paoletti, Dr. David Kritchevsky PDF

By Rodolfo Paoletti, Dr. David Kritchevsky

ISBN-10: 0120249146

ISBN-13: 9780120249145

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Possible involvement in the rhythm of activity modulation by hormones thus cannot at present be excluded from consideration. Although in its early stages, work on the effects of hormones on reductase activity is progressing rapidly. Evidence indicates that insulin and thyroid hormone may participate in the chain of events that initiate and maintain the increased rate of reductase synthesis that characterizes the rising portion of the rhythm (Lakshmanan et al, 1973; Huber et al, 1973a; Nepokroeff et al, 1974), while hydrocortisone and glucagon, possibly via cAMP, may b e involved in the chain of events that terminate reductase synthesis (Lakshmanan et al, 1973; Nepokroeff et al, 1974) (see also Section VIII).

This would not, however, preclude a significant change in the cholesterol content of an organelle such as the nucleus. On the other hand, changes in endogenous cholesterol synthesis may be sufficient to maintain a constant cholesterol level in liver in spite of changes in dietary input. , 1972). These inhibitors are nondialyzable and are relatively heat stable. 4 Id — T — i i ! 2 fei t! ) -à 26 28 L_ 30 AGE (DAYS) 85 of early and late weaning on rat hepatic HMG-CoA reductase activF I G . 16. Effect ity.

Reductase activity rises to a level slightly above normal, which persists for at least an additional 32 hours (Guder et al, 1968). , 1974). Two days after administration of triiodothyronine to hypophysectomized rats, reductase activity rises to 3-4 times the normal diurnal peak value, then falls to the initial, low activity (Fig. 20). , 1974) (Table IV). , 1973). These data suggest that triiodothyronine stimulation of reductase activity in normal animals may be limited by a negative control element, possibly hydrocortisone or glucagon.

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Advances in lipid research. / Volume 14 by Rodolfo Paoletti, Dr. David Kritchevsky

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